According to the well-known Coulomb failure criterion the variation of either stress or pore pressure can result in earthquake rupture. Aftershock sequences characterized by the Omori law are often assumed to be the consequence of varying stress, whereas earthquake swarms are thought to be triggered by fluid intrusions. The role of stress triggering can be analyzed by modeling solely three-dimensional (3-D) elastic stress changes in the crust, but fluid flows which initiate seismicity cannot be investigated without considering complex seismicity patterns resulting from both pore pressure variations and earthquake-connected stress field changes. We show that the epidemic-type aftershock sequence (ETAS) model is an appropriate tool to extract the primary fluid signal from such complex seismicity patterns. We analyze a large earthquake swarm that occurred in 2000 in Vogtland/NW Bohemia, central Europe. By fitting the stochastic ETAS model, we find that stress triggering is dominant in creating the observed seismicity patterns and explains the observed fractal interevent time distribution. External forcing, identified with pore pressure changes due to fluid intrusion, is found to directly trigger only a few percent of the total activity. However, temporal deconvolution indicates that a pronounced fluid signal initiated the swarm. These results are confirmed by our analogous investigation of model simulations in which earthquakes are triggered by fluid intrusion as well as stress transfers on a fault plane embedded in a 3-D elastic half-space. The deconvolution procedure based on the ETAS model is able to reveal the underlying pore pressure variations.